Here is an abstract from a 2015 publication on which a "K. Kersten" is second author. It mentions a "domino" effect:

Coffelt et al. (2015), "IL-17-producing γδ T cells and neutrophils conspire to promote breast cancer metastasis"

http://www.nature.com/nature/journal/v522/n7556/full/nature14282.html

The studies were done in "mouse models of spontaneous breast cancer metastasis", and they report""

"We mechanistically demonstrate that interleukin (IL)-1β elicits IL-17 expression from gamma delta (γδ) T cells, resulting in systemic, granulocyte colony-stimulating factor (G-CSF)-dependent expansion and polarization of neutrophils in mice bearing mammary tumours. Tumour-induced neutrophils acquire the ability to suppress cytotoxic T lymphocytes carrying the CD8 antigen, which limit the establishment of metastases. Neutralization of IL-17 or G-CSF and absence of γδ T cells prevents neutrophil accumulation and downregulates the T-cell-suppressive phenotype of neutrophils. Moreover, the absence of γδ T cells or neutrophils profoundly reduces pulmonary and lymph node metastases without influencing primary tumour progression. Our data indicate that targeting this novel cancer-cell-initiated domino effect within the immune system—the γδ T cell/IL-17/neutrophil axis—represents a new strategy to inhibit metastatic disease."

See also, this helpful Research Highlight piece regarding the above article (free full text):

Fridlender (2015), "Promoting metastasis: neutrophils and T cells join forces"

http://www.nature.com/cr/journal/v25/n7/full/cr201562a.html

For a broader review, see this article:

Welte (2015), "Interleukin-17 Could Promote Breast Cancer Progression at Several Stages of the Disease"

https://www.hindawi.com/journals/mi/2015/804347/

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